Last edited by Kigashakar
Monday, August 30, 2021 | History

4 edition of The kidney in mineralocorticoid-induced hypertension found in the catalog.

The kidney in mineralocorticoid-induced hypertension

Louie Mavrogiannis

The kidney in mineralocorticoid-induced hypertension

mechanisms for regulating blood prorenin and renin.

by Louie Mavrogiannis

  • 355 Want to read
  • 1 Currently reading

Published by National Library of Canada in Ottawa .
Written in English


Edition Notes

Thesis (M.Sc.) -- University of Toronto, 1994.

SeriesCanadian theses = -- Thèses canadiennes
The Physical Object
FormatMicroform
Pagination2 microfiches : negative. --
ID Numbers
Open LibraryOL17042108M
ISBN 100315963476
OCLC/WorldCa222158840


Share this book
You might also like
Acts and laws, passed by the Great and General Court or Assembly of His Majestys province of the Massachusetts-Bay, in New-England

Acts and laws, passed by the Great and General Court or Assembly of His Majestys province of the Massachusetts-Bay, in New-England

Solid fuel installations by a committee convened

Solid fuel installations by a committee convened

Templetons way with money

Templetons way with money

Prester John of the Indies

Prester John of the Indies

MCQs on diagnostic imaging

MCQs on diagnostic imaging

Cheetahs (Nature Watch (Carolrhoda Paperback))

Cheetahs (Nature Watch (Carolrhoda Paperback))

The kidney in mineralocorticoid-induced hypertension by Louie Mavrogiannis Download PDF EPUB FB2

As the most important mineralocorticoid, aldosterone is regulated by changes in sodium and potassium homeostasis and is also influenced by adrenocorticotropic hormone. Short title: SGK1 in mineralocorticoid-induced hypertension and kidney injury Corresponding author: Prof.

Diego Alvarez de la Rosa Departamento de Ciencias Médicas Author: Catalina Sierra-Ramos, Silvia Velazquez-Garcia, Ayse G. Keskus, Arianna Vastola-Mascolo, Ana E. Rodr. The kidneys were enlarged, and proteinuria was dramatically accelerated. All the above-mentioned changes induced by a high-salt challenge were attenuated by Cited by: 8.

Summary. To gain insight into the mechanism of mineralocorticoid-induced hypertension in man, we performed hemodynamic studies on six normotensive volunteers before Cited by: It is difficult, considering the volume of literature available on the subject, to present a brief synopsis of the kidney and hypertension.

The crux of the matter Author: E. Wardle. Hypertension Secrets, 2nd Edition, offers practical, up-to-date coverage of the full range of essential topics in this dynamic field.

This highly regarded resource. The mineralocorticoid receptor (MR) was first identified as a blood pressure regulator, modulating renal sodium handling in response to its principal ligand Cited by:   Init was proposed that cortisol was the mineralocorticoid that induced hypertension 5.

Biochemical findings were in agreement with this theory because in Cited by: P. Fuller, M. Young, in Hormones, Brain and Behavior (Second Edition), The mineralocorticoid receptor (MR) responds not only to the physiological.

Also, hypertension is associated with more cardiovascular disease deaths than any other modifiable disease conditions (Danaei et al.). Hypertension is a major. Introduction. Aldosterone secreted from the adrenal cortex increases the reabsorption of sodium and water by activating epithelial sodium channels (ENaCs) in the Cited by: The immunohistochemistry of kidney injury molecule 1, mineralocorticoid receptor and angiotensin converting enzyme were also determined.

Results indicated. Aldosterone, a steroid hormone secreted by the zona glomerulosa of the adrenal, has two principal functions: to prevent hyperkalemia and to protect against sodium : J. Howard Pratt. Hypertension is a frequent finding in both acute and chronic kidney disease, particularly with glomerular or vascular disorders [1] The pathogenesis and preferred.

Aldosterone, Volumethe latest release in the Vitamins and Hormones series first published incovers the field of hormone action, vitamin action, X-ray. Hypertension is a leading contributor to cardiovascular mortality worldwide. Despite this, its underlying mechanism(s) and the role of excess salt in cardiorenal Cited by: Request PDF | Apparent mineralocorticoid excess | Apparent mineralocorticoid excess (AME) is a potentially fatal genetic disorder causing severe juvenile.

The aldosterone-induced increases in striatin expression are mediated via activation of the mineralocorticoid receptor (MR) in cells. (a) cells. corticoid hypertension and congenital deficiency of 11 beta-hydroxysteroid dehydrogenase in a family with the syndrome of apparent mineralocorticoid excess.

The possibility that central mineralocorticoid binding sites are involved in the development of mineralocorticoid hypertension was examined using chronic blockade. Keywords:Spironolactone, aldosterone, proteinuria, chronic kidney disease, heart failure, hypertension, inflammation, fibrosis, renin-angiotensin aldosterone system.

Aldosterone-induced fibrosis in the kidney: questions and controversies. Brem AS (1), Morris DJ, Gong R. (1)Division of Kidney Diseases and Hypertension, Rhode Cited by:   Pitt B, Kober L, Ponikowski P, Gheorghiade M, Filippatos G, Krum H, et al.

Safety and tolerability of the novel non-steroidal mineralocorticoid receptor antagonist BAY. Etanercept also failed to decrease blood pressure in the deoxycorticosterone acetate (DOCA)-salt model of hypertension, although it was associated with a reduction in.

Chronic cold exposure causes hypertension and cardiac hypertrophy in rats within 13 wk (26, 27, 38, 39), namely cold-induced hypertension (CIH).

CIH is a natural Cited by:   The Kidney in Hypertension. Diana I. Jalal, Charles R. Nolan, and Robert W. Schrier. Global Burden of Hypertension.

Cardiovascular disease is the most common cause Estimated Reading Time: 12 mins. We hypothesized that combination treatment with the mineralocorticoid receptor antagonist eplerenone and the calcium channel blocker amlodipine elicits better. Arterial hypertension is a common condition worldwide and an important predictor of several complicated diseases.

Arterial hypertension can be triggered by many. Keywords:Aldosterone, eplerenone, spironolactone, mineralocorticoid receptor, mineralocorticoid receptor antagonists.

Abstract: Over the last decade a wealth of. A yr-old man presented with the classic signs of mineralocorticoid excess hypertension and hypokalemia.

The cause was not aldosterone excess, but elevation of. Two patients had a long history of hypertension and one of them also had a nephrotic-range proteinuria secondary to focal segmental scleronephrosis which had been.

Pathophysiology of chronic renal failure induced by hypertension Natural history of hypertension-induced renal damage The association between increased pulse.

Complications of HTNThe higher the BP and the longer anindividual has hypertension, the higherthe risk of complications which include: Hypertensive heart disease. Mineralocorticoid receptors (MR) and epithelial sodium channels (ENaC) in the brain mediate central aldosterone-induced sympathetic hyperactivity and hypertension.

Editor-in-Chief of the American Journal of Hypertension. By Paul J. Rosch, MD, F. The first part of this interview referred to a recent survey finding that of. Velvie A. Pogue is a nephrologist in Collierville, Tennessee. She received her medical degree from Harvard Medical School and has been in practice for more than 20.

Author: File Size: MB Format: PDF, ePub Download: Read: Scribd is the world's largest social reading and publishing site. - Read online for free. Tentang. Read free for 30 days.

Disclaimer This material is for educational purposes only. It is not to be used to make medical decisions. Medical decis.

Mineralocorticoids (e.g., aldosterone) support chronic inflammatory tissue damage, including glomerular mesangial injury leading to glomerulosclerosis. Furthermore. Dissertations / Theses on the topic 'STEROID-INDUCED HYPERTENSION' To see the other types of publications on this topic, follow the link: STEROID-INDUCED .You are leaving Medscape Education Cancel Continue.

For You.